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" Treatment of cerebral infarction : "
Jiro Suzuki
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BL
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| Record Number
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724890
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b544609
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| Main Entry
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Jiro Suzuki
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| Title & Author
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Treatment of cerebral infarction : : experimental and clinical study.\ Jiro Suzuki
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| Publication Statement
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[Place of publication not identified]: Springer, 2013
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| ISBN
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3709188636
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: 9783709188637
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| Contents
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I Experimental Study.- 1. Experimental Models.- 1 1 Introduction.- 1.2 Cerebral Infarction Models.- 1.2.1 Experimental Models Using the Monkey.- 1.2.2 Experimental Models Using the Dog.- 1.2.3 Experimental Models Using the Cat.- 1.2.4 Experimental Models Using the Rat.- 1.2.5 Experimental Models Using the Gerbil.- 1.3 The Cerebral Infarction Model Developed in Sendai.- 1.3.1 Production of Various Cerebral Infarction Models in the Dog by Means of Occlusion of Intracranial Trunk Arteries.- 1.3.1.1 Thalamic Infarction Model.- 1.3.1.2 Cerebral Mantle Infarction Model.- 1.3.1.3 Complete Cerebral Hemisphere Infarction Model.- 1.3.1.4 Incomplete Cerebral Hemisphere Infarction Model.- 1.3.1.5 Complete Ischemic Brain Regulated with the Perfusion Method.- 1.3.2 A 3-Vessel Occlusion Model for Bilateral Hemispheric Infarction Using the Rat.- 1.3.2.1 Production of the Ischemia-Model.- 1.3.2.2 EEG Studies.- 1.3.2.3 Studies of the Cerebral Blood Flow Using Autoradiography.- 2. Histological Study.- 2.1 Introduction.- 2.2 Sequential Changes in Cerebral Ischemia.- 2.2.1 Sequential Changes in Focal Ischemia.- 2.2.1.1 Acute Stage Changes in an Ischemic Focus.- 2.2.1.2 Changes in the Ischemic Focus Following Acute Stage Recirculation.- 2.2.1.3 Changes in Ischemic Foci During the Subacute and Chronic Stages.- 2.2.2 Sequential Changes in Global Ischemia.- 2.2.2.1 Observations Using the Light Microscope.- 2.2.2.2 Observations Using the Electron Microscope.- 2.2.2.3 Observation Using the Freeze Fracture Method.- 3. Cerebral Blood Flow.- 3.1 Introduction.- 3.2 Correlation Between Regional EEG and rCBF.- 3.3 Regional Cerebral Hemodynamics Following Recirculation.- 3.3.1 Hemodynamics at the Center of an Ischemic Focus.- 3.3.2 Hemodynamics of the Border and Periphery of an Ischemic Focus.- 3.4 Sequential Changes in Vascular Reactivities.- 3.4.1 Autoregulation.- 3.4.1.1 Sequential Change in Autoregulation Occurring at the Center of an Ischemic Focus.- 3.4.1.2 Sequential Changes in Autoregulation at the Border and in the Periphery of an Ischemic Focus.- 3.4.2 CO2 Response.- 3.5 Hemorrhagic Infarction.- 3.5.1 Conditions Conductive to Hemorrhagic Infarction.- 3.5.2 Hemodynamics Following Recirculation.- 4. Ischemic Brain Edema.- 4.1 Introduction.- 4.1.1 The Definition of Ischemic Brain Edema.- 4.1.2 Pathophysiology of Ischemic Brain Edema.- 4.1.2.1 Causal Factors.- 4.1.2.2 The Onset and Natural Course of Ischemic Brain Edema.- 4.1.2.3 Ischemic Brain Edema Following Recirculation.- 4.1.2.4 The Disappearance of Edema.- 4.1.3 The Treatment of Ischemic Brain Edema.- 4.1.3.1 General Therapeutic Steps.- 4.1.3.2 Drug Therapy.- 5. Cerebral Metabolism and Free Radical Pathology.- 5.1 Introduction.- 5.2 Phospholipids as Biomembrane Constituents.- 5.3 Ca2+ Homeostasis.- 5.4 Lipid Metabolism in Cerebral Ischemia.- 5.5 The Free Radical Reaction in Cerebral Ischemia.- 5.5.1 Fundamental Knowledge Concerning Free Radicals.- 5.5.2 Lipid Peroxidation.- 5.5.3 Detection of Free Radicals.- 5.5.3.1 Chemiluminescence.- 5.5.3.2 Electron Spin Resonance (ESR).- 5.5.4 Initiators of Autoxidative Lipid Peroxidation.- 5.5.4.1 Superoxide Anion
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| LC Classification
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RC394.I5J576 2013
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| Added Entry
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Jiro Suzuki
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