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" Essential Hypertension 2 "
edited by Kyuzo Aoki.
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BL
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| Record Number
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739236
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b559158
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| Main Entry
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edited by Kyuzo Aoki.
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| Title & Author
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Essential Hypertension 2\ edited by Kyuzo Aoki.
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| Publication Statement
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Tokyo: Springer Japan, 1989
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| Page. NO
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(xii, 404 pages 164 illustrations)
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| ISBN
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443168090X
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: 4431700447
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: 9784431680901
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: 9784431700449
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| Contents
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I Introduction: Spontaneously Hypertensive Rats and Classification of Hypertension --;Discovery and Development of the Spontaneously Hypertensive Rat --;Three-Way Classification of Hypertension: Gene Hypertension, Environment Hypertension, and Disease Hypertension --;II Calcium Movement During Contraction and Relaxation in Arterial Smooth Muscle --;Membrane Potential and Calcium Channels in Arterial Smooth Muscle Cells --;Excitatory Agonists and Ca-Permeable Channels in Arterial Smooth Muscle Cells --;Arterial Smooth Muscle Contraction Is Dependent on Sustained Increases in Myoplasmic Ca2+ --;Role of Second Messengers on Mobilization of Calcium and Related Events in Vascular Smooth Muscles --;Myogenic Contraction and Relaxation of Arterial Smooth Muscle --;Relationship Between Cytosolic Calcium Level and Contractile Tension in Vascular Smooth Muscle --;Calcium Mobilization Mechanisms in Smooth Muscle --;Sarcoplasmic Reticulum of Mesenteric Arteries Buffers Stimulated Ca2+ Entry --;Contraction Induced by Ouabain and Potassium-free Solution in Human Umbilical Arteries --;Pharmacology of Calcium Antagonists in Arterial Smooth Muscle --;III Membrane Calcium-Handling Abnormalities of Vascular Smooth Muscle in Spontaneously Hypertensive Rats --;Increased Calcium Permeability of the Membrane of Vascular Smooth Muscle in Spontaneously Hypertensive Rats --;Membrane Potential and Calcium Influx in Vascular Muscle from Spontaneously Hypertensive Rats --;Cell Membrane Properties of the Arterial Smooth Muscle from Spontaneously Hypertensive Rats --;Decreased?1-Adrenoceptor Reserve in Arteries from Spontaneously Hypertensive Rats --;Diminished?-Adrenoceptor-Mediated Relaxation of Arteries from Spontaneously Hypertensive Rats --;Changes of Vascular?- Adrenoceptors in Spontaneously Hypertensive Rats: Characterization and Technical Problems --;Vascular Mechanisms in Development and Maintenance of Hypertension in Spontaneously Hypertensive Rats --;Effects of Hypertension on Endothelial-Smooth Muscle Cell Interactions in Pressurized Cerebral Arteries --;Protective Effects of Calcium Antagonists on Hypertensive Diseases in Heart, Brain, and Kidney of Hypertensive Rats --;Hemodynamic and Hormonal Responses to Stimuli in Spontaneously Hypertensive Rats: Effects of Chronic Diltiazem Treatment --;Mechanism and Consequences of Cellular Calcium Elevation in Hypertension --;IV Calcium, Magnesium, and Calcium Antagonists in Human Hypertension --;Pressor Effects of Calcium Infusion in the Absence and Presence of a Calcium Antagonist in Subjects with Normotension and Hypertension --;Effects of Intravenous Infusion of Magnesium on Hemodynamics in Normotensives and Hypertensives --;Marked Blood Pressure Responses to Norepinephrine, Epinephrine, and Angiotensin II in Borderline Hypertension with a Parental History of Hypertension --;Responsiveness to Calcium Antagonists in Essential Hypertension: Pharmacokinetic and Neurohumoral Aspects --;Hemodynamic Effects of Calcium Antagonists at Rest and During Exercise in Essential Hypertension: A Comparison Between Verapamil, Diltiazem, Tiapamil, Nifedipine and Nisoldipine --;V Mechanism of Blood Plessure Elevation in Gene (Essential) Hypertension --;Calcium Metabolism in Hypertension: Clinical Evidence and Cellular Hypothesis --;The History of the Calcium Membrane Theory of Gene (Essential) Hypertension --;Index of Key Words.
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| Abstract
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Since Langendorff's discovery of the role of calcium ions in muscle contraction in 1895, we have made immense progress in our knowledge of the role of the calcium ion in the excitation-contraction coupling of the arterial smooth mus- cle.
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| Subject
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Cardiology.
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Medicine.
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| LC Classification
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RC685.H8E358 1989
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| Added Entry
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Kyuzo Aoki
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